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Erratum: TMEM140 is associated with the prognosis of glioma by promoting cell viability and invasion

The Original Article was published on 22 July 2015

It has been brought to our attention that in Fig. two of the article [1], the immunoblots in the middle panel were incorrectly labeled as TREM2 instead of TMEM140. The correct figure is included below (Fig. 1), in which the immunoblots in the middle panel are labeled as TMEM140. This labeling correction does not affect the results or interpretation of the results. We apologize for this error.

Fig. 1
figure 1

Suppressing of TMEM140 expression by RNAi. a TMEM140 expression level in five glioma cell lines was analyzed by RT-PCR (left) and immunoblot (middle and right). b, c The effect of TMEM140 knockdown through siRNA silencing. The cells were transfected with normal control or TMEM140-RNAi for 48 h and then subjected to RT-PCR (left) and immunoblot analysis (middle and right) of the TMEM140 expression level. The representative images for immunoblot are shown in the middle panel, and data from three independent experiments were expressed as the mean ± S.D. (right panel). Wild type: wild-type cells; normal control: scrambled siRNA transfected cells; RNAi-1, RNAi-2, and RNAi-3: TMEM140-RNAi-1, -2, and -3 transfected cells (*P < 0.05, **P < 0.01, ***P < 0.001 compared with normal control)

Reference

  1. Li B, Huang MZ, Wang XQ, Tao BB, Zhong J, Wang XH, et al. TMEM140 is associated with the prognosis of glioma by promoting cell viability and invasion. J Hematol Oncol. 2015;8(1):89. doi:10.1186/s13045-015-0187-4.

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Correspondence to Xiao-Qiang Wang or Shi-Ting Li.

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Bin Li and Ming-Zhu Huang contributed equally to this work.

The online version of the original article can be found under doi:10.1186/s13045-015-0187-4.

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Li, B., Huang, MZ., Wang, XQ. et al. Erratum: TMEM140 is associated with the prognosis of glioma by promoting cell viability and invasion. J Hematol Oncol 8, 101 (2015). https://doi.org/10.1186/s13045-015-0199-0

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  • DOI: https://doi.org/10.1186/s13045-015-0199-0